EC Number   |
General Information |
Reference |
|---|
   2.1.1.72 | malfunction |
a relevant defect of Yersinia enterocolitica Dam overproducers is enhanced invasion capacity, probably associated with transcriptional alterations in invasin genes inv and ail, and with changes in the composition of lipopolysaccharide O-antigen |
703765 |
| 2.1.1.72 | malfunction |
Dam negative mutants contain lowered levels of the secretion of pathogenicity island 1 transcriptional regulators HilA, HilC, HilD, and InvF. Dam negative mutants with the intestinal epithelium have been tentatively correlated with reduced secretion of pathogenicity island 1 |
703875 |
| 2.1.1.72 | malfunction |
deletion of the methyltransferase M.HpyAXII is lethal when associated with an active endonuclease, M.HpyAXII MTase activity is not required for establishing infection in mice |
-, 694457 |
| 2.1.1.72 | malfunction |
enzyme deletion suppresses the trans-generational phenotypes of spr-5 mutant worms |
742235 |
| 2.1.1.72 | malfunction |
enzyme gene disruption mutants fail to complete the sexual cycle when induced to mate and display complete lethality |
-, 756367 |
| 2.1.1.72 | malfunction |
enzyme loss-of-function leads to severe defects in growth and development |
-, 757856 |
| 2.1.1.72 | malfunction |
inactivation of the enzyme results in poor growth |
719450 |
| 2.1.1.72 | malfunction |
knockout of a putative DNA methyltransferase gene in Campylobacter jejuni causes reduced motility, aberrant flagellar appearance, and hyperadherence to epithelial cells, accompanied by reduced invasion |
703765 |
| 2.1.1.72 | malfunction |
lack of Dam methylation causes partial attenuation upon intranasal or intraperitoneal inoculation of mice |
703765 |
| 2.1.1.72 | malfunction |
mutants lacking Dam methylase are not viable, Dam overproduction does not impair bacterial growth |
703765 |
