EC Number   |
Expression |
Reference |
|---|
    2.3.2.2 | up |
at pH 4.0, the bacterium augments enzyme production in the presence of glutamine (about 10 mM) in the medium, which predominantly occurs after a 6-min time-lag |
-, 736623 |
| 2.3.2.2 | up |
exposure to cadmium increases the activities of both gammaglutamyl transferase and glutathione transferase 2 |
703704 |
| 2.3.2.2 | up |
isoform GGT2 expression is enhanced in ggt1 knockout mutants, suggesting a compensatory effect to restore enzyme activity in the root apoplast. Supplementation with 0.1 mM glutathione results in the 4fold up-regulation of isoform GGT2 gene expression |
720135 |
| 2.3.2.2 | up |
the VvGGT3 transcript in whole berries is upregulated upon Botrytis cinerea infection as well as mechanical damage to leaf tissue. In comparison to Botrytis infection, there is only a mild about 2.0fold increase in the relative transcript abundance of VvGGT3in mechanically wounded leaves compared to the unwounded control leaves at 6 h post wounding beacoming significantly different after 12 h |
756810 |
| 2.3.2.2 | up |
tumor necrosis factor TNFalpha induces GGT promoter transactivation, mRNA and protein synthesis, as well as enzymatic activity. Remicade, a clinically used anti-TNFalpha antibody, small interfering RNA against p50 and p65 nuclear factor NF-kB isoforms, curcumin, a well characterized natural NF-kB inhibitor, as well as a dominant negative inhibitor IkBa, prevent GGT activation at various levels. Over-expression of receptor of TNFalpha-1, TNFR-associated factor-2 TRAF2, TNFR-1 associated death domain TRADD, dominant negative IkBa or NF-kB p65 further confirm GGT promoter activation via NF-kB.Mutation of the NF-kB site located at 110 additionally inhibits TNFalpha-induced promoter induction |
702160 |
